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  • Winter 2006 - Issue Number 65
  • Winter 2006 - Issue Number 65

Below are the feature articles from Physiology News Issue 65, Winter 2006.

To read the full articles please download the issue.

  • Features

Functional domains of tropomyosin: new insights

Functional studies of tropomyosin molecule using transgenic approaches reveal that its inner core region has a greater effect on calcium sensitivity of the myofilaments and the carboxy terminal ends regulate the TM-actin affinity and/or crossbridge kinetics
  • Features

Legs pay out for cost of breathing!

Lee Romer and Jerome Dempsey propose that the demand placed on the respiratory muscles during high-intensity endurance exercise plays a pivotal role in determining exercise performance through its effect on limb blood flow and muscle fatigue
  • Features

Acute pain
  • Features

Endothelial nitric oxide synthase-derived NO signals regulate microvascular permeability

Regulation of vascular tone is largely dependent on the production of nitric oxide (NO) by the endothelium. Recent findings provide compelling evidence supporting an important role for endothelial NO in the regulation of microvascular permeability
  • Features

Organization of intermediary metabolism – lactate and its transporters
  • Features

Did evolution go the wrong path for the human lung?

Birds and mammals evolved along two separate evolutionary lines from the primitive reptiles. Although the physiology of many of the organ systems of birds and mammals are similar, the lungs are radically different. The thesis here is that the bird ended up with a better lung

  • Features

Acidification protects skeletal muscle volume during anaerobic exercise

Biophysical measurements and theoretical modelling of resting skeletal muscle exposed to extracellular lactate demonstrate that intracellular buffering of hydrogen ions precisely offsets the effect of increased intracellular lactate on cell volume

  • Features

What structures bear the tension and store energy in lengthening muscle?

When active muscles are stretched the cross-bridge cycle is truncated by suppressing the power stroke and thereby reducing ATP consumption; the high tension they exert is generated by stretch-induced strain in the pre-stroke cross-bridges. Other, non­crossbridge, structures also make a substantial contribution to tension and energy storage
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